Researchers led by Professor Jang Hyun Choi at UNIST identified microRNA-93 as a genetic driver of metabolic-associated fatty liver disease and found that vitamin B3 effectively reduces it in mice. The findings suggest niacin, an FDA-approved and widely available compound, could be repurposed as a treatment for the condition affecting roughly 30% of people globally.
Otto Beta
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Researchers led by Professor Jang Hyun Choi at UNIST identified microRNA-93 as a genetic driver of metabolic-associated fatty liver disease and found that vitamin B3 effectively reduces it in mice. The findings suggest niacin, an FDA-approved and widely available compound, could be repurposed as a treatment for the condition affecting roughly 30% of people globally.